103
MATERIAL SAFETY DATA SHEET Page : 3/4
Date Revised : Jul. 31, 2002
Date Issued : Jun. 1, 1998
MSDS No. F-00831
8.EXPOSURE CONTROLS / PERSONAL PROTECTION
Engineering Measures
Ventilation : Not required under intended use.
Exposure Limit Values
OSHA-PEL(USA) : 15mg/m3 (Total Dust), 5mg/m3 (Respirable Dust)
ACGIH-TLV(USA) : 10mg/m3 (Total Dust), 3mg/m3 (Respirable Dust)
Personal Protective Equipment
Respiratory Protection : Not required under intended use.
Hand Protection : Not required under intended use.
Eye Protection : Not required under intended use.
Skin Protection : Not required under intended use.
Other Protective Equipment : Use of a dust mask and goggles are recommended when handling a large
quantity of toner or during long term exposure, as with any non-toxic dust.
9.PHYSICAL AND CHEMICAL PROPERTIES
Appearance
Physical State : Solid Form : Powder Color : Black Odor : odorless
Ph : Not applicable
Boiling / Melting Point : Not applicable
Softening Point(°C) : No data
Flash Point(°C) : Not applicable
lgnition Point(°C ) : No data
Explosion Properties : No data
Density(g/cm³) : 1.1 (bulk density : 0.34)
Solubility in Water : Negligible
10.STABILITY AND REACTIVITY
Stability : Stable
Hazardous Reactions : Dust explosion, like most finely divided organic powders.
Conditions to Avoid : Electric discharge, throwing into fire.
Materials to Avoid : Oxidizing Materials
Hazardous Decomposition Products : CO, CO2 and NOX
Further Information : None
11.TOXICOLOGICAL INFORMATION
Acute Toxicity
Ingestion(oral) : LD50 > 2000mg/kg (Rats)
Dermal : LD50 > 2000mg/kg (Rats)
Inhalation : No Data
Eye irritation : Not an irritant
Skin irritation : Not an irritant
Skin sensitizer : No sensitization
Mutagenicity : Negative (Ames Test)
Carcinogenicity : In 1996 the IARC reevaluated carbon b lack as a Group 2B carcinogen (possible human
carcinogen). This classification is given to chemic als for which there is inadequate human
evidence, but sufficient animal evidence on which t o base an opinion of carcinogenicity. The
classification is based upon the development of lung tum ors in rats receiving chronic inhalation
exposures to free carbon black at levels that induce particle overload of the lung.
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